Fatty acid with less than six carbon atoms.
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1No significant differences in weight gain or stool SCFA content were detected.
2The production of SCFA increased with the level of supplied NT equivalents.
3SCFA levels were significantly higher in portal vs. hepatic and peripheral blood.
4No significant differences in stool SCFA content were detected between groups.
5SCFA had no significant effect on serum insulin or c-peptide concentrations.
6This was associated with an altered fecal SCFA and BA profile.
7In addition, the data provide indirect evidence for utilization of SCFA for lipid synthesis.
8The fall in FFA suggests that colonic SCFA have an effect on carbohydrate metabolism.
9However, the mechanisms underlying SCFA regulation of T-cell differentiation and function remain to be investigated.
10The ratios of SCFA formed differed depending on the microbial species involved in mucin cross-feeding.
11Analytical data on faecal samples support the role of the colonic microbial population in SCFA production.
12The data provide strong evidence that colonic SCFA are absorbed and metabolized in the human subject.
13In all groups SCFA concentrations were highest in the proximal colon and decreased towards the rectum.
14Increased melanoidins was found to result in significantly divergent gut microbiota profiles and supported sustained SCFA production.
15The main SCFA are acetate, propionate and butyrate which have numerous documented effects promoting large bowel function.
16Dietary stevia leave and stevioside decreased total concentration of SCFA and changed their profile in the ceca.